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Randall W. Burt, James S. Barthel, Kelli Bullard Dunn, Donald S. David, Ernesto Drelichman, James M. Ford, Francis M. Giardiello, Stephen B. Gruber, Amy L. Halverson, Stanley R. Hamilton, Mohammad K. Ismail, Kory Jasperson, Audrey J. Lazenby, Patrick M. Lynch, Edward W. Martin Jr., Robert J. Mayer, Reid M. Ness, Dawn Provenzale, M. Sambasiva Rao, Moshe Shike, Gideon Steinbach, Jonathan P. Terdiman and David Weinberg

includes well-defined inherited syndromes, such as Lynch syndrome (HNPCC), familial adenomatous polyposis (FAP), and MYH-associated polyposis (MAP). Other entities that are important to recognize include suspected colon cancer syndromes, such as Muir

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Brandie Heald, Shanna Gustafson, Jessica Mester, Patricia Arscott, Katherine Lynch, Jessica Moline and Charis Eng

counselor-only (GCO) clinics (A) and medical genetics clinics (B). Abbreviations: Fam Cancer, familial cancer; FAP/MAP, familial adenomatous polyposis or MYH-associated polyposis; GC/MD, genetic counselor and medical geneticist visit; HBOC, hereditary

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Patrick M. Lynch

possible FAP Summarize appropriate criteria for tumor testing for possible Lynch syndrome in patients with colorectal cancer Discuss the potential role of a universal testing strategy (UTS) for Lynch syndrome The 2013 NCCN Clinical Practice

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Heather Hampel

histology. Those with hamartomatous polyps may be found to have Peutz-Jeghers syndrome, juvenile polyposis, Cowden syndrome, or serrated polyposis syndrome; and those with adenomatous polyps may have familial adenomatous polyposis (FAP), attenuated FAP, or

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Patrick M. Lynch

. Limitations of the evidentiary base for extracolonic screening are conceded, with some cautious suggestions for possible strategies notwithstanding this lack of data. Advances in chemoprevention have been made in both familial adenomatous polyposis (FAP

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Margaret von Mehren, R. Lor Randall, Robert S. Benjamin, Sarah Boles, Marilyn M. Bui, Ephraim S. Casper, Ernest U. Conrad III, Thomas F. DeLaney, Kristen N. Ganjoo, Suzanne George, Ricardo J. Gonzalez, Martin J. Heslin, John M. Kane III, Joel Mayerson, Sean V. McGarry, Christian Meyer, Richard J. O’Donnell, Alberto S. Pappo, I. Benjamin Paz, John D. Pfeifer, Richard F. Riedel, Scott Schuetze, Karen D. Schupak, Herbert S. Schwartz, Brian A. Van Tine, Jeffrey D. Wayne, Mary Anne Bergman and Hema Sundar

, respectively. The mean age at diagnosis for rhabdomyosarcomas was younger than 6 years and the mean age at diagnosis was older than 50 years for undifferentiated pleomorphic sarcomas. Familial adenomatous polyposis (FAP) is an inherited autosomal

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Shilpa Grover and Sapna Syngal

polyposis (FAP), MYH -associated polyposis (MAP), and rare hamartomatous polyposis syndromes. This article focuses on the diagnostic features, evaluation, and management of Lynch syndrome, and includes a brief discussion of other inherited CRC syndromes

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Wylie Burke and Nancy Press

polyposis . Available at : http://www.geneclinics.org/servlet/access?db=geneclinics&site=gt&id=8888891&key=4vpwoPBUz29Za&gry=&fcn=y&fw=WCkt&filename=/profiles/fap/index.html . Accessed December 2005 . 29. Points to consider: ethical, legal

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Margaret von Mehren, R. Lor Randall, Robert S. Benjamin, Sarah Boles, Marilyn M. Bui, Ernest U. Conrad III, Kristen N. Ganjoo, Suzanne George, Ricardo J. Gonzalez, Martin J. Heslin, John M. Kane III, Henry Koon, Joel Mayerson, Martin McCarter, Sean V. McGarry, Christian Meyer, Richard J. O'Donnell, Alberto S. Pappo, I. Benjamin Paz, Ivy A. Petersen, John D. Pfeifer, Richard F. Riedel, Scott Schuetze, Karen D. Schupak, Herbert S. Schwartz, William D. Tap, Jeffrey D. Wayne, Mary Anne Bergman and Jillian Scavone

patients, respectively. The mean age at diagnosis for RMS was younger than 6 years, and the mean age at diagnosis for undifferentiated pleomorphic sarcomas was older than 50 years. Familial adenomatous polyposis (FAP) is an inherited autosomal

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Samir Gupta, Dawn Provenzale, Scott E. Regenbogen, Heather Hampel, Thomas P. Slavin Jr, Michael J. Hall, Xavier Llor, Daniel C. Chung, Dennis J. Ahnen, Travis Bray, Gregory Cooper, Dayna S. Early, James M. Ford, Francis M. Giardiello, William Grady, Amy L. Halverson, Stanley R. Hamilton, Jason B. Klapman, David W. Larson, Audrey J. Lazenby, Patrick M. Lynch, Arnold J. Markowitz, Robert J. Mayer, Reid M. Ness, Niloy Jewel Samadder, Moshe Shike, Shajanpeter Sugandha, Jennifer M. Weiss, Mary A. Dwyer and Ndiya Ogba

for CRC and management of genes with well-established risk for CRC, including Lynch syndrome, familial adenomatous polyposis (FAP), and MUTYH -associated polyposis (MAP), have been reviewed in detail in prior NCCN Guidelines, and have been updated