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Anne M. Covey

areas, development of cirrhosis in patients who were infected with hepatitis C virus (HCV) in the 1970s and 1980s, as well as the increase in nonalcoholic fatty liver disease. 2 As Dr. Covey pointed out, the incidence of disease parallels that of

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Muhammed Aasim Yusuf, Vinay Kumar Kapoor, Refaat Refaat Kamel, Ather Kazmi, Najam Uddin, Nehal Masood, and Abdulmajeed Al-Abdulkareem

. Raza SA Clifford GM Franceschi S . Worldwide variation in the relative importance of hepatitis B and hepatitis C viruses in hepatocellular carcinoma: a systematic review . Br J Cancer 2007 ; 96 : 1127 – 1134 . 6. Randi G Franceschi S La

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Presenter: Robin Kate Kelley

Conference. 1 “Its impact is not to be understated.” Most hepatocellular carcinomas (HCCs) are caused by an underlying liver injury due to toxins, such as alcohol, or viruses, such as hepatitis B virus (HBV) or hepatitis C virus (HCV). “But a very

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Amit G. Singal, Jorge A. Marrero, and Adam Yopp

of nonalcoholic fatty liver disease (NAFLD) and hepatitis C virus (HCV) cases. 1 Prognosis for patients with HCC depends on tumor stage at diagnosis, with curative options only available for patients diagnosed at an early stage. 2 , 3 Patients with

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Al B. Benson III, Thomas A. Abrams, Edgar Ben-Josef, P. Mark Bloomston, Jean F. Botha, Bryan M. Clary, Anne Covey, Steven A. Curley, Michael I. D'Angelica, Rene Davila, William D. Ensminger, John F. Gibbs, Daniel Laheru, Mokenge P. Malafa, Jorge Marrero, Steven G. Meranze, Sean J. Mulvihill, James O. Park, James A. Posey, Jasgit Sachdev, Riad Salem, Elin R. Sigurdson, Constantinos Sofocleous, Jean-Nicolas Vauthey, Alan P. Venook, Laura Williams Goff, Yun Yen, and Andrew X. Zhu

hepatic iron and alcohol ingestion in hepatic mutagenesis . Toxicology 2008 ; 254 : 11 – 18 . 26 Singal AK Anand BS . Mechanisms of synergy between alcohol and hepatitis C virus . J Clin Gastroenterol 2007 ; 41 : 761 – 772 . 27

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Nishant Patel, Adam C. Yopp, and Amit G. Singal

fatty liver disease (NAFLD) and hepatitis C virus (HCV). 1 Prognosis for patients with HCC depends on tumor stage at diagnosis, with curative options only available for those diagnosed at an early stage. Patients with early HCC achieve 5-year survival

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Nikolaos A. Trikalinos, Amy Zhou, Maria B. Majella Doyle, Kathryn J. Fowler, Ashley Morton, Neeta Vachharajani, Manik Amin, Jesse W. Keller, William C. Chapman, Elizabeth M. Brunt, and Benjamin R. Tan

an additional 33.3% patients were overweight (BMI, 25–30 kg/m 2 ). Less than 35% had a diagnosis of cirrhosis based on pathology and imaging review. A total of 21 patients were hepatitis C virus (HCV)–positive and 9 were hepatitis B virus (HBV

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Amit G. Singal, Akbar K. Waljee, Nishant Patel, Emerson Y. Chen, Jasmin A. Tiro, Jorge A. Marrero, and Adam C. Yopp

). Patients were classified according to origin of liver disease, including hepatitis C virus (HCV), hepatitis B virus, alcohol-related liver disease, and nonalcoholic fatty liver disease (NAFLD). Laboratory data of interest at time of diagnosis and treatment

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Lindsey Robert Baden, Sankar Swaminathan, Michael Angarone, Gayle Blouin, Bernard C. Camins, Corey Casper, Brenda Cooper, Erik R. Dubberke, Ashley Morris Engemann, Alison G. Freifeld, John N. Greene, James I. Ito, Daniel R. Kaul, Mark E. Lustberg, Jose G. Montoya, Ken Rolston, Gowri Satyanarayana, Brahm Segal, Susan K. Seo, Shmuel Shoham, Randy Taplitz, Jeffrey Topal, John W. Wilson, Karin G. Hoffmann, and Courtney Smith

transaminase (ALT) levels; patients with a history of injection drug use; men who have sex with other men; and patients positive for hepatitis C virus (HCV) or HIV. A positive HBsAg is associated with active infection or a window period before the development

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Ali Bazarbachi, Hamdy A. Azim, Hussain Alizadeh, Mahmoud Aljurf, Ibrahim Barista, Naeem A. Chaudhri, Zahira Fahed, Omar A. Fahmy, Ardeshir Ghavamzadeh, Mohamed H. Khalaf, Sami Khatib, Aghiad Kutoubi, Semra Paydas, Hanadi Rafii Elayoubi, Ghazi Zaatari, Hamdy M. Zawam, and Andrew D. Zelenetz

emergence of infectious diseases, such as hepatitis C virus infections. These malignant lymphomas represent a heterogeneous group of lymphoproliferative malignancies with differing patterns of behavior and responses to treatment. Most non-Hodgkin lymphoma